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Search Results to Tarun Garg

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One or more keywords matched the following properties of Garg, Tarun

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research overview My research is focus on developing natural killer (NK) cells for immunotherapeutic use against multiple myeloma, which has poor outcome despite novel therapeutic agents. In addition, elucidation of resistance mechanisms in myeloma is another area of my interest. Myeloma cells has the ability to evade immune surveillance through several potential mechanisms. Exploration of these mechanisms may lead to develop better therapeutic option in multiple myeloma.

One or more keywords matched the following items that are connected to Garg, Tarun

Item TypeName
Academic Article Bortezomib down-regulates the cell-surface expression of HLA class I and enhances natural killer cell-mediated lysis of myeloma.
Academic Article Highly activated and expanded natural killer cells for multiple myeloma immunotherapy.
Academic Article Infusion of haplo-identical killer immunoglobulin-like receptor ligand mismatched NK cells for relapsed myeloma in the setting of autologous stem cell transplantation.
Academic Article Combinatorial efficacy of anti-CS1 monoclonal antibody elotuzumab (HuLuc63) and bortezomib against multiple myeloma.
Academic Article Epigenetic modulation of MAGE-A3 antigen expression in multiple myeloma following treatment with the demethylation agent 5-azacitidine and the histone deacetlyase inhibitor MGCD0103.
Concept Multiple Myeloma
Academic Article Artesunate overcomes drug resistance in multiple myeloma by inducing mitochondrial stress and non-caspase apoptosis.
Academic Article Ex vivo-expanded natural killer cells demonstrate robust proliferation in vivo in high-risk relapsed multiple myeloma patients.
Grant Potentiating Natural Killer Cell Anti-Myeloma Effects
Grant Multiple Myeloma Cells Modulate ICAM-3 to Evade Natural Killer Cell-Mediated Lysis
Academic Article CST6 suppresses osteolytic bone disease in multiple myeloma by blocking osteoclast differentiation.
Grant Multiple Myeloma Cells Modulate ICAM-3 to Evade Natural Killer Cell-Mediated Lysis

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  • Multiple Myeloma